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specific effect as only fibrillar species of ataxin-3 showed binding. Furthermore, when the polyQ-binding peptide QBP1 was added there was no change to the binding pattern which suggests that binding occurs through the Josephin domain. This is similarly seen in the SDS experiments in this study, where the effect of SDS on the Josephin domain is identical to that on ataxin-3, and unaffected by QBP
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Cytoskeleton Location In Animal Cell

Posted by grey3weasel (#2233) 11 hours ago (Editorial)
highly expressed tyrosine kinase and cytokine signaling genes in primary human patient myeloma, we previously identified mutations at codon 12 and 61 in N- and KRAS as being the only recurrent variation in our sample set [4]. Recent genome sequencing efforts also found Ras mutations to be the most common single nucleotide variant (SNV) in MM [4], suggesting that Ras activation is an important eve
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